Herpetic Eye Disease: HSV & HZO Antiviral Therapy
A practical framework for herpetic eye disease
Antiviral management is driven by two questions: which virus (HSV vs. varicella-zoster) and which layer is involved (epithelium vs. stromal / endothelial / uveitic disease). HSV epithelial keratitis is treated to stop active viral replication and close the epithelial defect. In contrast, HSV stromal or endothelial disease is largely immune-mediated and often requires topical steroid with concurrent antiviral coverage to reduce the risk of epithelial reactivation.
Herpes zoster ophthalmicus (HZO) requires prompt systemic antiviral therapy, ideally started early after rash onset, to reduce ocular complications and help mitigate post-herpetic neuralgia. Topical antivirals usually have a limited role in HZO compared with HSV epithelial disease.
HSV epithelial keratitis (dendritic and geographic ulcers)
The classic presentation is a dendritic ulcer: a branching epithelial defect with terminal bulbs that stains with fluorescein, often with rose bengal or lissamine staining of devitalized epithelium. Treatment goals are to halt viral replication, promote re-epithelialization, and reduce progression to a geographic ulcer or stromal involvement.
Topical antiviral therapy: Ganciclovir 0.15% gel is commonly favored for HSV epithelial disease due to good tolerability and convenient dosing. Continue through epithelial closure, then taper per response and clinical protocol.
Oral antiviral therapy: Oral acyclovir, valacyclovir, or famciclovir are effective options and avoid additional surface toxicity. Selection is individualized based on age, renal function, adherence considerations, and local practice patterns. Dose and duration should follow current labeling and institutional guidance.
Avoid topical steroids in active epithelial HSV disease (dendritic/geographic ulcers), as steroids can promote viral replication and enlarge epithelial defects.
HSV stromal, endothelial, and uveitic disease
Stromal keratitis (necrotizing or immune stromal), endothelial involvement (disciform keratitis), and HSV-associated anterior uveitis are typically driven by inflammation and immune response. These phenotypes often require a carefully monitored topical steroid to control inflammation and limit scarring, but they should be paired with concurrent antiviral coverage (commonly oral) to reduce the risk of epithelial reactivation.
Management decisions (timing, taper, duration) depend on severity, corneal findings, IOP, recurrence history, and follow-up reliability. Co-management is appropriate when disease is severe, atypical, recurrent, or threatening the visual axis.
Herpes zoster ophthalmicus (HZO)
HZO involves the ophthalmic (V1) distribution of the trigeminal nerve and requires systemic antiviral therapy to reduce viral replication and ocular complications. Benefit is greatest when therapy starts early after rash onset. Unlike HSV epithelial disease, topical antivirals are not routinely central to HZO care; treatment is typically high-dose oral antiviral therapy tailored to renal function, age, and comorbid conditions. Coordinate with primary care, urgent care, or neurology for severe pain, systemic involvement, or complex cases.